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29 Sentences With "environmental pollutant"

How to use environmental pollutant in a sentence? Find typical usage patterns (collocations)/phrases/context for "environmental pollutant" and check conjugation/comparative form for "environmental pollutant". Mastering all the usages of "environmental pollutant" from sentence examples published by news publications.

This is concerning because artificial light is an environmental pollutant.
"The issue was simply whether carbon was an environmental pollutant or not," he said.
It's a long-term environmental pollutant that serves as a "raft that shuttles other pollutant in our water, food and bodies," Halden said.
It also might be the only place in England where curry is categorized not just as an entree, but also as an environmental pollutant.
They include companies tackling drug discovery using digital twin cells, environmental pollutant monitoring, and artificial brain development for robotics applications, to name just a few (that last one was a mouthful).
It is an environmental pollutant and is toxic to fish.
2,4,6-Trichlorophenol is an environmental pollutant that has been found in fresh water lakes such as the Great Lakes.
E. Monosson and C. Cleveland (eds.). Washington DC. but a cadmium-dependent carbonic anhydrase has been found in some marine diatoms. Cadmium is considered an environmental pollutant that causes health hazard to living organisms.Xu, Liang, Zhang, Fei, Tang, Mingjia, et al.
Diethanolamine is another such compound, but also an environmental pollutant. N-cyclohexylcholine inhibits choline uptake primarily in brains. Hemicholinium-3 is a more general inhibitor, but also moderately inhibits choline kinases. More specific choline kinase inhibitors have also been developed.
Cadmium is a heavy metal and an environmental pollutant. Cadmium is a known carcinogen. Cadmium exposure has been linked to lung, prostate and renal cancer. It is not a strong mutagen, but acts as a promoter through mitogenic effects on gene expression.
Lead paint on a porch in 2018 Lead-based paint was widely used in the United States, because of its durability. The United States banned the manufacture of lead-based house paint in 1978 due to health concerns. Lead has long been considered to be a harmful environmental pollutant. Cited cases of lead poisoning date back to the early 20th century.
The polymeric materials that tires they are made of do not decompose easily. Even after heavy use and wear, only a few grams are abraded from each tire before they are deemed not serviceable. This means that almost all the material is discarded, and a valuable resource is left to become an environmental pollutant, if dumped.Adhikari, B., De, D., & Maiti, S. (2000).
Use of polymer matrix syntactic foams in USS Zumwalt for lightweight and stealth has been reported. Gupta worked on the use of fly ash hollow particles (cenospheres) in creating syntactic foams. Fly ash is an environmental pollutant and beneficial uses of this material are desired. The work of fly ash utilization in composite materials was featured in National Geographic and Fast Company magazine.
Conventional bleaching of wood pulp using elemental chlorine produces and releases into the environment large amounts of chlorinated organic compounds, including chlorinated dioxins. Pdf Dioxins are recognized as a persistent environmental pollutant, regulated internationally by the Stockholm Convention on Persistent Organic Pollutants. Dioxins are highly toxic, and health effects on humans include reproductive, developmental, immune and hormonal problems. They are known to be carcinogenic.
OPR has shown to also function in the reduction of explosive 2,4,6-trinitrotoluene (TNT). Because TNT is a known toxic, environmental pollutant that is difficult to degrade, the use of phytoremediation to clean up sites contaminated with TNT is of significant interest. OPR1 degraded TNT faster and with greater amount of degraded products than other isozymes. This enzyme could therefore be used in phytoremediation.
The efficiency and efficacy of each method of remediation has limitations. The goal of remediation is to eliminate the environmental pollutant as quickly as possible; only inefficient processes require human intervention. Environmental factors such as requirements of reaction, mobility of substances, and physiological needs of organisms will affect the rate and degree that contaminants are degraded. Over time, many of these requirements are overcome.
Panellus stipticus has been shown to reduce the phenolic concentration of waste water produced by olive-processing plants—an environmental concern in many Mediterranean countries. In this study, a liquid culture of P. stipticus mycelia reduced the initial concentration of phenolic compounds by 42% after a 31-day incubation period. In a separate study, a P. stipticus culture was able to effectively degrade the environmental pollutant 2,7-dichlorodibenzo-p-dioxin, a polychlorinated dioxin.
Balkhashtsvetmet is the strongest environmental pollutant in the region of the Lake Balkhash. The release of wastewater from the combine into the lake leads to numerous diseases of the fauna. Industrial untreated waste water was discharged into the aquatic environment of the lake since the launch of the combine in 1938 through 1995. As a result, according to the fisheries, the contamination of components of water and air sector with heavy metals has become dominant in the present day.
Research has shown that the environmental pollutant methylmercury can increase the prevalence of homosexual behavior in male American white ibis. The study involved exposing chicks in varying dosages to the chemical and measuring the degree of homosexual behavior in adulthood. The results discovered was that as the dosage was increased the likelihood of homosexual behavior also increased. The endocrine blocking feature of mercury has been suggested as a possible cause of sexual disruption in other bird species.
Some methylotrophs within the genus of Methylobacillus can even use organic compounds such as the pesticide carbofuran and choline as carbon raw materials; they use these carbon sources to fulfill their energy and carbon requirements. As early as the late 1980s researchers had known that some methylotrophs possess enzymes such as dichloromethane dehalogenase, or methane monooxygenase (MMO), which degrade various environmental pollutants (i.e.: alkanes, alkenes, and mono- and poly-substituted aromatic compounds). Another common environmental pollutant that results from industrial processes is formaldehyde.
At age 19Dreaming the Future: Reimagining Civilization in the Age of Nature, p.xvi Ausubel woke up one morning paralyzed on the left side of his body. Unable to see straight or even swallow, he went to several different doctors but none of them could diagnose what had caused the stroke-like symptoms. Out of desperation Ausubel sought alternative forms of medicine where he learned what had caused his health crisis- massive exposure to a chemical called dioxin, a toxic environmental pollutant.
Conventional bleaching using elemental chlorine produces and releases into the environment large amounts of chlorinated organic compounds, including chlorinated dioxins. Dioxins are recognized as a persistent environmental pollutant, regulated internationally by the Stockholm Convention on Persistent Organic Pollutants. Dioxins are highly toxic, and health effects on humans include reproductive, developmental, immune and hormonal problems. They are known to be carcinogenic. Over 90% of human exposure is through food, primarily meat, dairy, fish and shellfish, as dioxins accumulate in the food chain in the fatty tissue of animals.
Genetically modified fish (GM fish) are organisms from the taxonomic clade which includes the classes Agnatha (jawless fish), Chondrichthyes (cartilaginous fish) and Osteichthyes (bony fish) whose genetic material (DNA) has been altered using genetic engineering techniques. In most cases, the aim is to introduce a new trait to the fish which does not occur naturally in the species, i.e. transgenesis. GM fish are used in scientific research and kept as pets. They are being developed as environmental pollutant sentinels and for use in aquaculture food production.
Excess caesium can lead to hypokalemia, arrythmia, and acute cardiac arrest, but such amounts would not ordinarily be encountered in natural sources. As such, caesium is not a major chemical environmental pollutant. The median lethal dose (LD50) value for caesium chloride in mice is 2.3 g per kilogram, which is comparable to the LD50 values of potassium chloride and sodium chloride. Caesium chloride has been promoted as an alternative cancer therapy, but has been linked to the deaths of over 50 patients, on whom it was used as part of a scientifically unvalidated cancer treatment.
A collaborative study between the U.S. Environmental Protection Agency (EPA) and the University of New Hampshire investigated the effect of DE-71, a commercial PBDE mixture, on PEPCK enzyme kinetics and determined that in vivo treatment of the environmental pollutant compromises liver glucose and lipid metabolism possibly by activation of the pregnane xenobiotic receptor (PXR), and may influence whole-body insulin sensitivity. Researchers at Case Western Reserve University have discovered that overexpression of cytosolic PEPCK in skeletal muscle of mice causes them to be more active, more aggressive, and have longer lives than normal mice; see metabolic supermice.
Similar to other rodenticides, the preparation requires 3–7 days to be effective. Rather than killing rodents through internal haemorrhaging as anticoagulants do, PCC affects a rodent’s digestive system, causing acute dehydration due to its extremely absorptive nature (corn cob has been used in applications such as oil spills in water bodies, seed drying and de-icing). Physiological digestive pathways are disrupted, preventing normal regulation of water and salt levels, leading to hypovolemic circulatory shock through reduced blood volume and blood pressure, oxygen deprivation of the blood, and ultimately death. It is by nature biodegradable, thus not an environmental pollutant.
During her time at Duke, Bilbo investigated neuroimmune interactions in brain development, and the effect of neonatal infection on glial cell biology and immune functions later in life. In 2016, Bilbo joined the faculty at Harvard Medical School and became the Lurie Family Associate Professor of Pediatric and Neuroscience as well as the director of research for the Lurie Center for Autism at the Massachusetts General Hospital for Children. During this time she did research on the effect of adolescent exposure to morphine on long-term microglial gene expression. She also studied the effect of environmental pollutant exposure during critical periods of prenatal development on metabolic, behavioral, and neuroinflammatory developments in adult offspring.
The Mace Head station has been recording mercury (Hg) since 1995, and has thus been one of the longest running mercury recording stations in the world. Mercury data recording and collection is vital as it is a global environmental pollutant emitted both naturally and from man-made sources. Evidence suggests that mercury emissions from man made sources are as great as those originating from natural sources, thus it is the man-made emitted mercury that is contributing to an increase in environmental exposure to mercury. Long term and continuous measurements are necessary as it details whether a fluctuation is normal or something unusual, thus allowing one to monitor background levels of total gaseous mercury.
Shorter transcripts with missing exons or an alternative 3' terminal exon have been detected in various studies; however, their role in the cell is unclear. The 5' region of the NEDD9 promoter is regulated by all-trans retinoic acid (ATRA), and contains a retinoic acid response element (RARE) that is specifically bound by a retinoid X receptor (RXR)/retinoic acid receptor (RAR) heterodimer. NEDD9 is also induced by the environmental pollutant dioxin, based on regulation through the aryl hydrocarbon receptor (AhR). One study has found NEDD9 repressed by estrogen, based on binding of the SAFB1 co-repressor. NEDD9 is induced by Wnt signaling in colon cancer, based on binding to T-cell factor (TCF) factors in the promoter region. NEDD9 is induced by hypoxia and loss of VHL, based on binding of hypoxia-induced factor (HIF) transcription factors to the NEDD9 promoter. Prostaglandin E2 induces NEDD9 transcription. The Fox transcription factor Forkhead box C1 (FoxC1) and PAX5 transcription factor have been reported to induce NEDD9 transcription.

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